![]() Median sternotomy and circulatory arrest, Įarly Operative Results of IAA Mortality & morbidity 1) Early death Acute or subacute heart failure without or with multisystem failure.* Direct anastomosis (open or non-open technique).* Urgent operation is advisable, preferably.the absence of any deficit of CD4 T lymphocyte.* Facial feature with hypocalcemia and IAA in.* Pulmonary vascular disease after one year.* 1~4% of CHD in autopsy ( 1.3% of CHD in infancy ) Here, we report the first instance of a patient with 6q2122 deletion presenting with interrupted aortic arch in addition to the previously described clinical signs.* Closing PDA anuria & metabolic acidosis.* Critically ill neonate in severe CHF.- hypocalcemia & immunologic problem (Type B).* DiGeorge’s syndrome (absence of thymic tissue).* Truncus arteriosus, aortopulmonary window.* Rare interruption in right aortic arch.* Anomalies of brachiocephalic vessels are frequent.Associated cardiac anomalies are common, particularly a nonrestrictive VSD, and IAA comprises less than 1% of all CHD.Spontaneous ductal closure results in systemic hypoperfusion, metabolic acidosis, and end-organ failure.Luminal interruption between the ascending and descending aorta is found, and distal blood flow is dependent on a patent ductus arteriosus.Surgical correction, which must be performed in the infant, carries a higher risk. Truncus arteriosus, absent pulmonary valve syndrome, and interrupted aortic arch type B are the most serious defects. Celoria and Patton : A, B, C type in 1959 The presence of an aortic arch anomaly increases the odds of having a 22q11.2 deletion, regardless of the intracardiac anatomy.A complete luminal & anatomic discontinuity between.Interrupted Aortic Arch Seoul National University Hospital Department of Thoracic & Cardiovascular Surgery
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